For this assignment write a response to the issues addressed below. Be sure to cover all aspects of the questions. This assignment is to be typed, double-spaced, 12 pt font size, proper margins, and written in essay form. While I am primarily interested in the quality of your ideas, elements such as spelling, grammar, syntax, and form will be included in the overall assessment of this assignment. This essay is to be a MINIMUM of 3 FULL pages.
Based on your reading of the ideas/theories related to self-concept, in chapter three of our text and class lecture/discussion, respond to each of the following in essay form: 1) Analyze the primary sources that helped shape/influence your self-concept: significant others (parents, primary caregivers, early education teachers, etc.), cultural teachings, social comparison (peers, friends, colleagues), life experience. (Refer to the above model). 2) How do you perceive yourself in terms of: A. Your physical appearance B. Your belief systems (religion/philosophy), C. Your degree of self-esteem, D. Your social traits What are your strengths and weaknesses in each area? How do you think others perceive you in each area? How do you rate yourself in comparison to others in each area? What were your cultural teachings in each area?
3) How has your perception of self-influenced your perception of others?
4) What steps have you taken to increase perceptual accuracy when it comes to judging/evaluating/categorizing other people?
Presentation: Prostate malignant growth is the most widely recognized disease of men and the third most regular reason for death from malignancy. Prostate malignant growth as a rule happens in men over the age of 50. Numerous men who have prostate malignant growth, don’t demonstrate any indications. Screening for this malignant growth is generally done by a Digital rectum test (DRE) or potentially Prostate explicit antigen test (PSA). Advanced rectum test is the palpation of the prostate through the rear-end to check the span of the prostate. Prostate explicit antigen test is a blood test to check if the prostate is discharging this particular antigen in the circulatory system which can be a solid sign of prostate malignancy. Research has been done on avoiding and diminishing the danger of acquiring this disease. In these examinations, nourishment can assume an essential job in decreasing the odds of getting this malignant growth. Diets that high in foods grown from the ground, low in fat particularly fat from meats are appeared to diminish the hazard and advancement of prostate malignancy. Plant Based Diets: Explicit nutrition classes have appeared a few investigations to be a urgent device in counteracting and moderating the movement of this infection. One gathering of sustenances that have appeared critical decrease in staying away from prostate disease is plant based nourishments. Plant-based nourishments are appeared to contain high measures of nutrients and components known to lessen malignant growth cell. For instance, plant-based sustenance contains nutrients and phytochemicals, for example, carotenoids, flavonoids, phytoestrogens and isothiocyanates (Ma and Chapman, 2009). These phytochemicals have appeared to relapse disease cell improvement and stop tumor advancement. Besides plant based eating regimens are high in cancer prevention agent. Cell reinforcements are particles that keep the oxidation of different atoms. Oxidation is the purpose for the generation of free radicals. Likewise, cancer prevention agents decrease the harm to DNA by authoritative to these free radicals and dispensing with them from the body. Free radicals harm the structure of DNA which can prompt tumor development. A vital carotenoid and phytochemical is lycopene. Lycopene is found in natural products, for example, tomatoes. Lycopene is known to decrease insulin-like development factor 1 (IGF-1) marking. IGF-1 is protein that has found to advance prostatic malignancy cells and enable the prostate to be powerless to disease. Cruciferous vegetables such cauliflower, broccoli, Brussels grows and bok choy all contain a compound call glucosinolate. Glucosinolate have appeared in concentrates to diminish DNA harm, initiate apoptosis and hinder prostate malignant growth cell (Ma and Chapman, 2009).They additionally have phenethyl isothiocyanate, sulfophorane and indole-3-carbinol, which are exacerbates that have potential enemy of disease properties. Their system of activity includes the hindrance of cytochrome P450 catalysts, which oxidize mixes, for example, benzo[a]pyrene and polycyclic fragrant hydrocarbons (PAHs) into increasingly polar epoxy-diols. Cytochrome P450 chemical is a realized operator to cause change and disease development Another vegetable that establishes an effect on tumor cells is soy bean. Soy beans contain isoflavones*. Isoflavones are natural 3-phenylchromen-4-one structure substances that regular happen in various sorts of sustenances. They hinder catalysts related with transmission of signs for tumor cell development. ‘Tea particularly green has been found to have comparative impacts to expending vegetables and natural products. Polyphenol mixes in green tea have likewise been found to have indistinguishable impacts from the cruciferous vegetables. They counteract metastases of the malignancy, instigate apoptosis and restrain cell development. Studies have appeared green tea restrains tumor development, in this way, prompting lower prostate-explicit antigen levels and lower IGF-1 levels (Ma and Chapman, 2009). One of these polyphenolic mixes is epigallocathechin-3 gallate (EGCG). EGCG is known to connect to urokinase, Urokinase is a chemical that enables tumor cells to develop. In the present market, there is a medication called amiloride and is utilized Amiloride can just have a most extreme portion of 20 mg EGCG while ordinary measure of green can have upwards of 150mg EGCG. Another advantage is that green tea can be devoured at a lot higher rate than amiloride and not have physiological impact on the body (Jankun ,1997, p561) . Fish Oil The following gatherings of nourishment that have appeared to hinder tumor cell development are fish oils. Fish oil contains n3 polyunsaturated unsaturated fats. n3 polyunsaturated unsaturated fats are known to decrease serum prostate-explicit antigen (PSA) protein articulation (Ma and Chapman, 2009). PSA is delivered by the prostate organ and is utilized by the body to condense semen. The body utilizes discharges PSA in semen however in some cases within the sight of malignancy, the PSA getaways to the circulatory system. There two primary gatherings of unsaturated fats, omega 3 and omega 6 unsaturated fats. , Omega 3 unsaturated fats shield from malignant growth, while omega 6 unsaturated fats have been found to advance disease when devoured in extreme sums. One motivation behind why omega 6 advances malignant growth is a direct result of its capacity to advancement aggravation. (Tandon et al., 2008). Nutrients and Minerals Another cancer prevention agent that has demonstrated critical impact on prostate malignancy cell development is nutrient E. Nutrient E is considered as an intracellular cancer prevention agent and an antiprostaglandin. * Antiprostaglandins are a gathering of particles that lessen the generation of middle people of the incendiary procedure. Similarly as with any fat dissolvable nutrients, an excess of can have hurtful impact. Admission of more than 400 IU can be unsafe to the body (Ma and Chapman, 2009). Moreover another nutrient that is critical in repressing tumor cell development is nutrient D. In studies, nutrient d has appeared both ordinary cells and dangerous to change over nutrient d into a functioning hormonal state. In this state, nutrient D has appeared to restrain tumor become particularly prostatic epithelial cells. (Tandon et al., 2008). Another substance other than nutrients that can help in the battle against is minerals. Minerals vary from nutrients in that nutrients are natural and contain carbon while minerals don’t and are think about inorganic. A specific mineral, selenium, has been turned out to be a defensive specialist. It diminishes cell multiplication, causes apoptosis and restrains angiogenesis. (Mama and Chapman, 2009). Angiogenesis is the development of new fine veins in the body. In forestalling vein arrangement; the tumor can’t develop or spread (Li, Smith and Li, 2011). Perils of Meats, high fat weight control plans and Nitrites Every one of the nourishments examined above have appeared to diminish the case of prostatic malignant growth cells. Then again, there are gatherings of sustenance that are presently known to advance prostate disease cells. For instance, meats particularly cooked at high temperatures add to the creation of malignant growth cells. At the point when a meat is cook at high temperature, it discharges cancer-causing agents. Cancer-causing agents are known to change DNA which can prompt two outcomes, apoptosis or advances uninhibited cell development. The two cancer-causing agents that are created amid this procedure are Heterocyclic amines (HCAs) and polycyclic sweet-smelling hydrocarbons (PAHs). (“Why Carcinogens Cause Cancer”, n.d.). An approach to decrease HCA and PAH development in cooked meats is to lessen the measure of time the meat is cooked on an open fire. This should be possible by persistently flipping the meat or utilizing a microwave to abbreviate the cooking time on the stove. Another cancer-causing agent that is found in meat is N-nitrosocompound. This cancer-causing agent is found in bacon, wieners and anything containing sodium nitrate. Sodium nitrate is additive added to nourishment to safeguard as well as to add flavor to. N-nitroso is framed when the sodium nitrate consolidates with the amines in the meat to shape this cancer-causing agent. These N-nitroso compound assaults cells and can make change the particle’s DNA (Mehdad, 2010). In another examination, there was proof that a high fat eating routine add to prostate malignant growth. In this examination, men shopper high sorts of monosaturated and polyunsaturated fats altogether increment their odds of getting prostate malignant growth (Crowe et al., 2008). Men who were expending 45 grams of all out fat in multi day have 10-20 percent expanded danger of getting disease (Park, Murphy, Wilkens, Henderson, and Kolonel, 2007). In another investigation, there was a solid connection among’s corpulence and prostate disease. In an investigation of eighty-seven men with prostate malignancy, 89 percent of these men were overweight or corpulent. Ninety-seven percent of them had a muscle to fat ratio of twenty five percent or higher (Mehdad, McBride, Grillo, Camilo, and Ravasco, 2010). Heftiness builds the body’s creation of IGF-1 which examined early prompts malignant growth cell multiplication. End Similarly as with anything, explicit gatherings of sustenances should be taken with some restraint. Every day action and a decent feast can contribute enormously to avoid malignant growth development as well as carrying on with a sound glad life. Prostate disease is the most well-known malignant growth for men and the second driving reason for disease demise in men. Eating nourishments high in cancer prevention agents, phytochemicals, for example, vegetables and natural products can prompt the decrease of prostatic malignant growth cell framing. What’s more, dodging consumes less calories high in protected meats, journal and cooking at high temperature all can furthermore lessen men’s odds of getting prostate disease. Works Cited Crowe, F.L., Key, T.J., Appleby, P.N., Travis, R.C., Overvad, K., Jakobsen, M.U., Riboli, E. (2008). Dietary fat admission and danger of prostate disease in the European Prospective Investigation into Cancer and Nutrition. The American Journal of Clinical Nutrition, 87: 1405-1413. Jankun, J. (1997, June 5). Why drinking green tea could forestall malignancy. Nature, 381, 561. Li, W., Hutnik, M., Smith, R., and Li, V. (2011). Understanding Angiogenesis. Recovered from http://www.angio.org/ua.php. April 4, 2014. Mama, R.W.- L. and Chapman, K. (2009). A precise survey of the impact of eating regimen in prostate malignant growth anticipation and treatment. Diary of Human Nutrition and Dietetics, 22:187-199. Mehdad, A., McBride, E., Grillo, I.M., Camilo, M., and Ravasco, P. (2010).Nutritional status and eat>