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Building A Career

compare the major publicly accessible career management websites and test their capabilities.

Founded in 1995, Careerbuilder.com claims to be the largest online job site in the United States. A chief rival is Monster.com, which pioneered digital recruitment in 1994. Its parent company, Monster Worldwide, Inc., also offers similar services in other countries with local listings. For more variety, feel free to use Linkedin.com or Dice.com in this assignment. All of these companies earn revenue from fees charged to employers for posting jobs and searching through resumes for qualified candidates, and also from online advertising.

Select any two career management websites to explore and compare your experience. As you create accounts and explore each site, take note of various features such as the interface and ease of use, resume building, search capability, job offerings, etc. Document your findings in a table format in a Word document.
In a paragraph below the table, explain which site you prefer and the reasons behind your choice.

Sample Solution

prokaryotes) [REF Behrman EJ, 2005 8] and derived via the mevalonate (MVA) pathway. The MVA is a fundamental metabolic network providing essential elements for normal cellular metabolism and executed in the endoplasmic reticulum (ER) and cytoplasm of a cell. Despite the presence of MVA pathway in almost all animal cells, the contribution per organ differs. The human brain generates vast amounts of de novo synthesized cholesterol, approximately 20% of the total cholesterol pool and primary FC, mainly found in myelin sheaths that insulate axons [REF dietschy turley 2004 9]. Moreover, the hepatic contribution to the cholesterol pool derived from de novo synthesis varies per species, hepatic cells in mice contribute approximately 40% to the whole cholesterol synthesis, while human liver cells adds only 10% to the total pool [REF Dietschy turley 2001 10 REF 30 Goedeke ]. The MVA-pathway is a highly controlled enzymatic process, resulting in the stepwise formation of FC [REF reviewed by 11 tricarico 2015 16067-16084]. The newly formed cellular cholesterol is either directly used as a precursor for metabolites (bile acids, steroids, water soluble vitamins, included in the membrane) or converted towards CE by acyl-Co A acyl transferase (ACAT) and either effluxed towards the plasma compartment or stored in lipid droplets [REF 12 35 goedeke]. The stored CE within lipid droplets can be converted into FC by hormone sensitive lipase (HSL)[REF]. Since appropriate cellular cholesterol levels are critical for normal cell metabolism, the regulation of intracellular cholesterol levels are tightly controlled by feedback mechanisms that operate at both transcriptional as well as post-transcriptional levels [REF goedeke 10.11]. Low cellular cholesterol triggers the MVA-pathway to upregulate the activation of the rate limiting enzymes i.a. 3-hydroxy-3methylgkutaryl (HMGCR) [REF] and receptor mediated exogenous uptake [REF]. High cellular cholesterol levels activate nuclear hormone receptors that in turn trigger transcription of cholesterol efflux related genes i.a. AB
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