Explain the value of invasive hemodynamic monitoring, including a discussion of whether an acute, chronic, or complex-care patient is a candidate for this type of assessment. For a complex-care patient, propose an evidence-based treatment plan regarding the hemodynamic information. What are the risk factors to take into consideration for this patient?
discuss the following invasive hemodynamic pressures to which you are assigned.
provide the normal values and discuss the differential diagnoses for the alteration in normal readings for systemic vascular resistance.
Support your summary and recommendations plan with a minimum of two APRN-approved scholarly resources.
The value of invasive hemodynamic monitoring
it can also be beneficial in chronic and complex care settings when patients exhibit hemodynamic instability or require specialized interventions.
Acute Care Patients:
Acute care patients are those with sudden, life-threatening illnesses or injuries. They often have unstable hemodynamics and require frequent assessment to identify and correct abnormalities promptly. IHM provides real-time hemodynamic data, enabling clinicians to make timely and informed treatment decisions.
Chronic Care Patients:
Chronic care patients have long-term illnesses, such as heart failure or chronic obstructive pulmonary disease (COPD). While their hemodynamics may be more stable than acute care patients, IHM can still be valuable in assessing the effectiveness of treatment, identifying early signs of worsening hemodynamics, and optimizing therapy to prevent complications.
Complex Care Patients:
Complex care patients have multiple medical problems or conditions, making their assessment challenging using traditional methods like physical examination and vital signs. IHM provides comprehensive hemodynamic data, aiding in understanding the patient's complex physiologic state and guiding appropriate treatment interventions.
Evidence-Based Treatment Plan for Complex Care Patients
An evidence-based treatment plan for a complex care patient with abnormal hemodynamics should be tailored to the patient's specific circumstances and hemodynamic profile. However, general principles include:
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Optimizing Preload: Preload is the volume of blood in the ventricles before they contract. Optimizing preload can be achieved through fluid administration, inotropes, or vasopressors.
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Optimizing Afterload: Afterload is the resistance against which the ventricles must pump. Optimizing afterload involves using vasodilators or beta-blockers.
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Optimizing Contractility: Contractility is the force of the heart's contraction. Optimizing contractility can be achieved using inotropes.
Risk Factors Associated with IHM
IHM, while beneficial, carries certain risks that should be carefully considered:
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Bleeding: IHM can increase the risk of bleeding, particularly in patients with anticoagulation or thrombocytopenia.
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Infection: IHM can increase the risk of infection, especially if catheters are not properly inserted and maintained.
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Arrhythmias: IHM can trigger arrhythmias, especially in patients with underlying heart disease.
Systemic Vascular Resistance (SVR)
Systemic vascular resistance (SVR) is a measure of the overall resistance to blood flow in the body. It is calculated as the difference between mean arterial pressure (MAP) and central venous pressure (CVP), divided by cardiac output (CO).
Normal values for SVR range from 700 to 1200 dynes/sec/cm^5.
Differential Diagnoses for Altered SVR
Deviations from the normal SVR range can indicate various underlying conditions.
Increased SVR:
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Vasoconstriction: Narrowing of blood vessels due to factors like sympathetic nervous system activation, pain, or certain medications.
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Increased Blood Volume: Fluid overload, kidney failure, or certain medications can increase blood volume, leading to elevated SVR.
Decreased SVR:
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Vasodilation: Widening of blood vessels due to parasympathetic nervous system activation, certain medications, or sepsis.
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Decreased Blood Volume: Dehydration, hemorrhage, or certain medications can decrease blood volume, resulting in lowered SVR.
Scholarly Resources
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Marik, P., & Vachon, F. (2016). Invasive hemodynamic monitoring in the intensive care unit. Critical Care Medicine, 44(12), e783.
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Rhodes, A., Grounds, M., Cecconi, M., & Vincent, J.-L. (2012). Hemodynamic monitoring and outcome in critical care. Chest, 141(6), 1338-1344.